Keap1-independent GSK-3β/Nrf2 signaling mediates electroacupuncture inhibition of oxidative stress to induce cerebral ischemia-reperfusion tolerance
EA exerts antioxidative stress effects against CIR injury by activating the GSK-3β/Nrf2 signaling pathway, independent of Keap1 regulation.
Authors
- Zhengde Wen (ORCID: https://orcid.org/0000-0002-8544-3438)
- Shan Luo (ORCID: https://orcid.org/0000-0003-3368-9935)
- Baojun Huang (ORCID: https://orcid.org/0000-0002-9855-1147)
- Chunjue Ni (ORCID: https://orcid.org/0000-0003-3112-472X)
- Yufan Huang
Institutions
- Wenzhou Medical University (CN)
- First Affiliated Hospital of Wenzhou Medical University (CN)
Publication Details
- Journal
- Brain Research Bulletin
- Published
- 2024-09-05
- DOI
- https://doi.org/10.1016/j.brainresbull.2024.111071
- Citations
- 8
- Primary Topic
- Healthcare and Venom Research
- Type
- article
- Field-Weighted Citation Impact
- 3.58
Funders
- Natural Science Foundation of Zhejiang Province