P-, E-, and H-cadherins differ in their relationships with coronary stenosis, cardiovascular outcomes, and unplanned recurrent revascularization

The data indicated that classical and atypical cadherins were associated with atherosclerosis progression. Elevated levels of P-cadherin were associated with coronary atherosclerosis. The data indicated that various lipid, endothelial, and metabolic biomarkers may influence the levels of cadherins. Thus, P-, E-, and H-cadherins may be promising markers for the assessment of cardiovascular risk.

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Publication Details

Journal
Journal of Molecular and Cellular Cardiology Plus
Published
2024-09-01
DOI
https://doi.org/10.1016/j.jmccpl.2024.100091
Citations
4
Primary Topic
Acute Myocardial Infarction Research
Type
article
Field-Weighted Citation Impact
1.61
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article

P-, E-, and H-cadherins differ in their relationships with coronary stenosis, cardiovascular outcomes, and unplanned recurrent revascularization

Yaroslav I. Havrichenko, Nadezhda G. Gumanova, Dmitry K. Vasilyev, Natalya L. Bogdanova et al.
4 citations
Journal of Molecular and Cellular Cardiology Plus
Acute Myocardial Infarction Research
1.61
article

P-, E-, and H-cadherins differ in their relationships with coronary stenosis, cardiovascular outcomes, and unplanned recurrent revascularization

Yaroslav I. Havrichenko, Nadezhda G. Gumanova, Dmitry K. Vasilyev, Natalya L. Bogdanova, Oxana M. Drapkina
article en
4 citations

Abstract

The data indicated that classical and atypical cadherins were associated with atherosclerosis progression. Elevated levels of P-cadherin were associated with coronary atherosclerosis. The data indicated that various lipid, endothelial, and metabolic biomarkers may influence the levels of cadherins. Thus, P-, E-, and H-cadherins may be promising markers for the assessment of cardiovascular risk.

Journal of Molecular and Cellular Cardiology PlusVol. 9
National Research Center for Preventive Medicine (RU)
Good health and well-being
Openalex Percentile: Top 15%
Acute Myocardial Infarction Research
1.61
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