Molecular mechanisms of PI3K isoform dependence in embryonic growth
These findings suggest that multiple cellular parameters, including PTEN status, PI3K isoform levels, and Rac1 activity, combine to influence PI3K isoform prevalence, rather than a single determinant.
Authors
- Onur Çizmecioğlu (ORCID: https://orcid.org/0000-0002-7608-6950)
- Sena Atıcı (ORCID: https://orcid.org/0000-0002-3532-3788)
Institutions
- Bilkent University (TR)
- University of Würzburg (DE)
Publication Details
- Journal
- Journal of the Turkish-German Gynecological Association
- Published
- 2024-08-29
- DOI
- https://doi.org/10.4274/jtgga.galenos.2024.2024-6-7
- Primary Topic
- PI3K/AKT/mTOR signaling in cancer
- Type
- article
- Field-Weighted Citation Impact
- 0.00