Association of Unc-51-like Kinase 4 ( <i>ULK4</i> ) with the reactivity of the extended reward system in response to conditioned stimuli
Our findings provide further evidence in humans that genetic variation in <i>ULK4</i> may increase the vulnerability to mental disorders, by modulating the extended reward system function. Future studies are needed to confirm the modulation of the extended reward system by <i>ULK4</i> and to specify the role of this mechanism in the pathogenesis of psychiatric disorders.
Authors
- Karolin E. Einenkel
- Oliver Gruber
- Esther K. Diekhof (ORCID: https://orcid.org/0000-0003-3826-8494)
- Rosanne Picotin
- Jens Treutlein (ORCID: https://orcid.org/0000-0003-3952-8905)
- Simone Löhlein
Institutions
- Universität Hamburg (DE)
- Heidelberg University (DE)
- Universität der Bundeswehr München (DE)
- University of Göttingen (DE)
Publication Details
- Journal
- The World Journal of Biological Psychiatry
- Published
- 2024-08-26
- DOI
- https://doi.org/10.1080/15622975.2024.2393381
- Citations
- 1
- Primary Topic
- Parkinson's Disease Mechanisms and Treatments
- Type
- article
- Field-Weighted Citation Impact
- 0.37