Enhanced T cell activation and cytotoxicity against AML via targeted anti-CD99 nanoparticle treatment

CD99 is a transmembrane protein overexpressed in Acute Myeloid Leukemia (AML), presenting a potential novel therapeutic target. Our group has previously developed anti-CD99-A192 (α-CD99-A192), comprising of single chain variable fragment (scFv) and elastin-like polypeptides (ELPs), and reported promising anti-leukemic activity in AML preclinical models. Treatment with α-CD99-A192 induced apoptosis in AML cell lines and prolonged survival in AML xenograft models. Considering CD99's expression and role in T cell activation, in the current study, we propose that α-CD99-A192 plays a dual function, i.e., targeting leukemic cells and activating T cells. This manuscript reports the effects of α-CD99-A192 on T cells in the context of AML. α-CD99-A192 treatment enhances T cell proliferation and activation and increases the release of pro-inflammatory cytokines along with increased aggregation of T cells, which culminates in heightened cytotoxicity against leukemic cells. Altogether, these findings suggest α-CD99-A192 enhances T cell activation and cytotoxic potential consistent with dual mechanisms of action for α-CD99-A192.

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Publication Details

Journal
Biomedicine & Pharmacotherapy
Published
2024-08-20
DOI
https://doi.org/10.1016/j.biopha.2024.117265
Citations
1
Primary Topic
Cell Adhesion Molecules Research
Type
article
Field-Weighted Citation Impact
0.38

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article

Enhanced T cell activation and cytotoxicity against AML via targeted anti-CD99 nanoparticle treatment

Houda Alachkar, J. Andrew MacKay, Yiting Meng, Mateusz Pospiech et al.
1 citations
Biomedicine & Pharmacotherapy
Cell Adhesion Molecules Research
0.38
article

Enhanced T cell activation and cytotoxicity against AML via targeted anti-CD99 nanoparticle treatment

Houda Alachkar, J. Andrew MacKay, Yiting Meng, Mateusz Pospiech, Atham Ali, Sandra Onyemaechi, Kanaka Dhuri, Shephali Kadam
article en
1 citations

Abstract

CD99 is a transmembrane protein overexpressed in Acute Myeloid Leukemia (AML), presenting a potential novel therapeutic target. Our group has previously developed anti-CD99-A192 (α-CD99-A192), comprising of single chain variable fragment (scFv) and elastin-like polypeptides (ELPs), and reported promising anti-leukemic activity in AML preclinical models. Treatment with α-CD99-A192 induced apoptosis in AML cell lines and prolonged survival in AML xenograft models. Considering CD99's expression and role in T cell activation, in the current study, we propose that α-CD99-A192 plays a dual function, i.e., targeting leukemic cells and activating T cells. This manuscript reports the effects of α-CD99-A192 on T cells in the context of AML. α-CD99-A192 treatment enhances T cell proliferation and activation and increases the release of pro-inflammatory cytokines along with increased aggregation of T cells, which culminates in heightened cytotoxicity against leukemic cells. Altogether, these findings suggest α-CD99-A192 enhances T cell activation and cytotoxic potential consistent with dual mechanisms of action for α-CD99-A192.

Biomedicine & PharmacotherapyVol. 179
University of Southern California (US)
University of Southern California, National Institutes of Health, National Cancer Institute
Good health and well-being
Openalex Percentile: Top 37%
Cell Adhesion Molecules Research
0.38
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Enhanced T cell activation and cytotoxicity against AML via targeted anti-CD99 nanoparticle treatment — Houda Alachkar, J. Andrew MacKay, et al. · Biomedicine & Pharmacotherapy (2024) | TGRS Research Map | TGRS