TMEM17 Promotes Tumor Progression in Glioblastoma by Activating the PI3K/AKT Pathway

These data prove that <i>TMEM17</i> plays a key role in the regulation of GBM and has great potential as a clinical therapeutic target for GBM.

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Publication Details

Journal
Frontiers in Bioscience-Landmark
Published
2024-08-16
DOI
https://doi.org/10.31083/j.fbl2908285
Citations
7
Primary Topic
Glioma Diagnosis and Treatment
Type
article
Field-Weighted Citation Impact
3.07

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article

TMEM17 Promotes Tumor Progression in Glioblastoma by Activating the PI3K/AKT Pathway

Quan Zhou, Chang Liu, Sheng Yan, Fujun Li et al.
7 citations
Frontiers in Bioscience-Landmark
Glioma Diagnosis and Treatment
3.07
article

TMEM17 Promotes Tumor Progression in Glioblastoma by Activating the PI3K/AKT Pathway

Quan Zhou, Chang Liu, Sheng Yan, Fujun Li, Maolin He, Daqin Feng, Shengyong Wang
article en
7 citations

Abstract

These data prove that <i>TMEM17</i> plays a key role in the regulation of GBM and has great potential as a clinical therapeutic target for GBM.

Frontiers in Bioscience-LandmarkVol. 29(8)
Guangxi Medical University (CN), First Affiliated Hospital of GuangXi Medical University (CN)
National Natural Science Foundation of China
Openalex Percentile: Top 8%
Glioma Diagnosis and Treatment
3.07
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