The Deubiquitinase USP22‐Stabilized COL17A1 Promotes Lung Adenocarcinoma Progression

Our findings demonstrate that USP22-stabilized COL17A1 possesses oncogenic activity in LUAD. We propose that USP22 and COL17A1 would be potential targets for the establishment of therapeutic approaches against LUAD.

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Publication Details

Journal
The Clinical Respiratory Journal
Published
2024-08-01
DOI
https://doi.org/10.1111/crj.13824
Citations
3
Primary Topic
Ubiquitin and proteasome pathways
Type
article
Field-Weighted Citation Impact
0.70
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article

The Deubiquitinase USP22‐Stabilized COL17A1 Promotes Lung Adenocarcinoma Progression

Huifeng Li, Dandan Du, Haihua Wang, Guangxi Chen
3 citations
The Clinical Respiratory Journal
Ubiquitin and proteasome pathways
0.70
article

The Deubiquitinase USP22‐Stabilized COL17A1 Promotes Lung Adenocarcinoma Progression

Huifeng Li, Dandan Du, Haihua Wang, Guangxi Chen
article en
3 citations

Abstract

Our findings demonstrate that USP22-stabilized COL17A1 possesses oncogenic activity in LUAD. We propose that USP22 and COL17A1 would be potential targets for the establishment of therapeutic approaches against LUAD.

The Clinical Respiratory JournalVol. 18(8)
Jiujiang First People's Hospital (CN)
Good health and well-being
Openalex Percentile: Top 30%
Ubiquitin and proteasome pathways
0.70
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The Deubiquitinase USP22‐Stabilized COL17A1 Promotes Lung Adenocarcinoma Progression — Huifeng Li, Dandan Du, et al. · The Clinical Respiratory Journal (2024) | TGRS Research Map | TGRS