Melatonin Improves Oxidative Stress Injury in Retinopathy of Prematurity by Targeting miR-23a-3p/Nrf2

Our study showed that melatonin could ameliorate H<sub>2</sub>O<sub>2</sub>-induced apoptosis and oxidative stress injury in RGC cells by mediating miR-23a-3p/Nrf2 signaling pathway, thereby improving retinal degeneration.

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Publication Details

Journal
Current Eye Research
Published
2024-08-05
DOI
https://doi.org/10.1080/02713683.2024.2380433
Primary Topic
Retinopathy of Prematurity Studies
Type
article
Field-Weighted Citation Impact
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article

Melatonin Improves Oxidative Stress Injury in Retinopathy of Prematurity by Targeting miR-23a-3p/Nrf2

Feng Zhang, Fei Tang, Zhixian Gou, Liqun Lu et al.
Current Eye Research
Retinopathy of Prematurity Studies
article

Melatonin Improves Oxidative Stress Injury in Retinopathy of Prematurity by Targeting miR-23a-3p/Nrf2

Feng Zhang, Fei Tang, Zhixian Gou, Liqun Lu, Fan Yang, Xuemei Ning, Yue Zhou
article en

Abstract

Our study showed that melatonin could ameliorate H<sub>2</sub>O<sub>2</sub>-induced apoptosis and oxidative stress injury in RGC cells by mediating miR-23a-3p/Nrf2 signaling pathway, thereby improving retinal degeneration.

Current Eye ResearchVol. 49(12)
Chengdu Medical College (CN), First Affiliated Hospital of Chengdu Medical College (CN)
Zero hunger
Openalex Percentile: Top 82%
Retinopathy of Prematurity Studies
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