KCTD17-mediated Ras stabilization promotes hepatocellular carcinoma progression
KCTD17 induces the stabilization of Ras and downstream signaling pathways and HCC progression and may represent a novel therapeutic target for HCC.
Authors
- Michele Carrer (ORCID: https://orcid.org/0000-0002-6464-9584)
- Junjie Yu (ORCID: https://orcid.org/0000-0001-5603-6094)
- Utpal B. Pajvani (ORCID: https://orcid.org/0000-0001-5991-2723)
- Kyung Hee Jung (ORCID: https://orcid.org/0000-0002-1291-0461)
- Young Hoon Jung (ORCID: https://orcid.org/0000-0002-0781-3608)
- Tam Dao (ORCID: https://orcid.org/0000-0001-6611-0497)
- Soon‐Sun Hong (ORCID: https://orcid.org/0000-0001-7679-1388)
- KyeongJin Kim (ORCID: https://orcid.org/0000-0001-6865-9141)
- Ah‐Reum Oh (ORCID: https://orcid.org/0000-0001-5783-0345)
- Dongryeol Ryu (ORCID: https://orcid.org/0000-0002-5335-6209)
- Sang Bae Lee (ORCID: https://orcid.org/0000-0002-4031-8051)
- Yun Ji Lee (ORCID: https://orcid.org/0000-0003-2702-7599)
- HyunJoon Gi
- Yelin Jeong
- Young Un Kim
Institutions
- China Pharmaceutical University (CN)
- Inha University (KR)
- Ionis Pharmaceuticals (United States) (US)
- Gwangju Institute of Science and Technology (KR)
- Columbia University (US)
- Jeonbuk National University (KR)
Publication Details
- Journal
- Clinical and Molecular Hepatology
- Published
- 2024-08-05
- DOI
- https://doi.org/10.3350/cmh.2024.0364
- Citations
- 9
- Primary Topic
- ATP Synthase and ATPases Research
- Type
- article
- Field-Weighted Citation Impact
- 2.10
Funders
- National Research Foundation
- Inha University
- National Research Foundation of Korea
- Ministry of Science and ICT, South Korea
- National Institutes of Health