Astragaloside IV induces endothelial progenitor cell angiogenesis in deep venous thrombosis through inactivation of PI3K/AKT signaling.

AS-IV prevents thrombus in an experimental DVT model by facilitating EPC angiogenesis and decreasing inflammation through inactivation of PI3K/AKT signaling.

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Publication Details

Journal
PubMed
Published
2024-09-01
DOI
https://doi.org/10.14670/hh-18-704
Citations
4
Primary Topic
Venous Thromboembolism Diagnosis and Management
Type
article
Field-Weighted Citation Impact
2.20
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article

Astragaloside IV induces endothelial progenitor cell angiogenesis in deep venous thrombosis through inactivation of PI3K/AKT signaling.

Xiaojiang Lyu, Zhigang Yi, Chonghai Liu, Chunfeng Zhang et al.
4 citations
PubMed
Venous Thromboembolism Diagnosis and Management
2.20
article

Astragaloside IV induces endothelial progenitor cell angiogenesis in deep venous thrombosis through inactivation of PI3K/AKT signaling.

Xiaojiang Lyu, Zhigang Yi, Chonghai Liu, Chunfeng Zhang, Yun He, Ping Zhu
article en
4 citations

Abstract

AS-IV prevents thrombus in an experimental DVT model by facilitating EPC angiogenesis and decreasing inflammation through inactivation of PI3K/AKT signaling.

PubMedVol. 39(9)
Affiliated Hospital of North Sichuan Medical College (CN)
Openalex Percentile: Top 15%
Venous Thromboembolism Diagnosis and Management
2.20
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